Diabetes

雜志介紹

Diabetes雜志介紹

《Diabetes》是一本以English為主的未開放獲取國際優秀期刊，中文名稱糖尿病，本刊主要出版、報道醫學-ENDOCRINOLOGY & METABOLISM領域的研究動態以及在該領域取得的各方面的經驗和科研成果，介紹該領域有關本專業的最新進展，探討行業發展的思路和方法，以促進學術信息交流，提高行業發展。該刊已被國際權威數據庫SCIE收錄，為該領域相關學科的發展起到了良好的推動作用，也得到了本專業人員的廣泛認可。該刊最新影響因子為6.2，最新CiteScore 指數為12.5。

本刊近期中國學者發表的論文主要有：

• Genetic Regulation of Pigment Epithelium-Derived Factor (PEDF): An Exome-Chip Association Analysis in Chinese Subjects With Type 2 Diabetes.

  Author: Cheung CYY^1,2, Lee CH¹, Tang CS³, Xu A^1,2,4,5, Au KW¹, Fong CHY¹, Ng KKK¹, Kwok KHM¹, Chow WS¹, Woo YC¹, Yuen MMA¹, Hai J¹, Tan KCB¹, Lam TH⁶, Tse HF^7,8, Sham PC^9,10,11, Lam KSL^7,2,4.

• Targeting the CDA1/CDA1BP1 Axis Retards Renal Fibrosis in Experimental Diabetic Nephropathy.

  Author: Chai Z¹, Wu T², Dai A², Huynh P², Koentgen F³, Krippner G⁴, Ren S^2,5, Cooper ME².

• HSPA12A Is a Novel Player in Nonalcoholic Steatohepatitis via Promoting Nuclear PKM2-Mediated M1 Macrophage Polarization.

  Author: Kong Q¹, Li N¹, Cheng H¹, Zhang X², Cao X¹, Qi T¹, Dai L¹, Zhang Z³, Chen X¹, Li C⁴, Li Y⁵, Xue B⁶, Fang L⁷, Liu L⁸, Ding Z⁹.

• Endoplasmic Reticulum-Associated Degradation (ERAD) Has a Critical Role in Supporting Glucose-Stimulated Insulin Secretion in Pancreatic β-Cells.

  Author: Hu Y¹, Gao Y¹, Zhang M¹, Deng KY², Singh R³, Tian Q¹, Gong Y¹, Pan Z¹, Liu Q¹, Boisclair YR³, Long Q⁴.

英文介紹

Diabetes雜志英文介紹

Diabetes publishes original research about the physiology and pathophysiology of diabetes mellitus. Submitted manuscripts can report any aspect of laboratory, animal, or human research. Emphasis is on investigative reports focusing on areas such as the pathogenesis of diabetes and its complications, normal and pathologic pancreatic islet function and intermediary metabolism, pharmacological mechanisms of drug and hormone action, and biochemical and molecular aspects of normal and abnormal biological processes. Studies in the areas of diabetes education or the application of accepted therapeutic and diagnostic approaches to patients with diabetes mellitus are not published.